Atherosclerosis

Lipid-free apolipoprotein (apo) A-I is converted into alpha-migrating high density lipoproteins by lipoprotein-depleted plasma of normolipidemic donors and apo A-I-deficient patients but not of Tangier disease patients.

Atherosclerosis

von Eckardstein A, Huang Y, Kastelein JJ, Geisel J, Real JT, Kuivenhoven JA, Miccoli R, Noseda G, Assmann G

Lipid-free apolipoprotein (apo) A-I is converted into alpha-migrating high density lipoproteins by lipoprotein-depleted plasma of normolipidemic donors and apo A-I-deficient patients but not of Tangier disease patients.

Atherosclerosis

von Eckardstein A, Huang Y, Kastelein JJ, Geisel J, Real JT, Kuivenhoven JA, Miccoli R, Noseda G, Assmann G

Diabetes mellitus in a new kindred with familial hypobetalipoproteinemia and an apolipoprotein B truncation (apoB-55).

Atherosclerosis

Pulai JI, Latour MA, Kwok PY, Schonfeld G

Diabetes mellitus in a new kindred with familial hypobetalipoproteinemia and an apolipoprotein B truncation (apoB-55).

Atherosclerosis

Pulai JI, Latour MA, Kwok PY, Schonfeld G

High-density lipoprotein cholesterol as a predictor of coronary heart disease risk. The PROCAM experience and pathophysiological implications for reverse cholesterol transport.

Atherosclerosis

Assmann G, Schulte H, von Eckardstein A, Huang Y

High-density lipoprotein cholesterol as a predictor of coronary heart disease risk. The PROCAM experience and pathophysiological implications for reverse cholesterol transport.

Atherosclerosis

Assmann G, Schulte H, von Eckardstein A, Huang Y

Determination of the fine specificity and molecular cloning of two monoclonal antibodies directed against the receptor-binding domain of human apolipoprotein E.

Atherosclerosis

R. Raffai¨, E. Rassart, K. Weisgraber, T. Innerarity, R. Milne

Determination of the fine specificity and molecular cloning of two monoclonal antibodies directed against the receptor-binding domain of human apolipoprotein E.

Atherosclerosis

R. Raffai¨,E. Rassart,K. Weisgraber,T. Innerarity,R. Milne

Lipopolysaccharide stimulation of RAW 264.7 macrophages induces lipid accumulation and foam cell formation.

Atherosclerosis

Funk JL, Feingold KR, Moser AH, Grunfeld C

Lipopolysaccharide stimulation of RAW 264.7 macrophages induces lipid accumulation and foam cell formation.

Atherosclerosis

Funk JL, Feingold KR, Moser AH, Grunfeld C

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